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The term allostasis denotes long-term adaptation processes, by which the organism maintains homeostasis in the context of chronic stressor load. This ensures survival, but also increases waste and ageing (allostatic load). Allostasis results from a directed change in the parameters of affected information processing structures. Consequently, beneficial alterations in the behaviour of the respective system ensue, but in the long-term also setpoint diseases. The term was first coined by Sterling and Eyer in order to sharpen the concept of stress response. Examples for allostatic reactions are

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  • Allostasis
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  • The term allostasis denotes long-term adaptation processes, by which the organism maintains homeostasis in the context of chronic stressor load. This ensures survival, but also increases waste and ageing (allostatic load). Allostasis results from a directed change in the parameters of affected information processing structures. Consequently, beneficial alterations in the behaviour of the respective system ensue, but in the long-term also setpoint diseases. The term was first coined by Sterling and Eyer in order to sharpen the concept of stress response. Examples for allostatic reactions are
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  • The term allostasis denotes long-term adaptation processes, by which the organism maintains homeostasis in the context of chronic stressor load. This ensures survival, but also increases waste and ageing (allostatic load). Allostasis results from a directed change in the parameters of affected information processing structures. Consequently, beneficial alterations in the behaviour of the respective system ensue, but in the long-term also setpoint diseases. The term was first coined by Sterling and Eyer in order to sharpen the concept of stress response. Examples for allostatic reactions are * Increased secretion of glucocorticoids in long-term stress situations * Hyperdeiodation (hyperdeiodination) and hypodeiodation (hypodeiodination) leading to central and peripheral components, respectively, of non-thyroidal illness syndrome * Incomplete compensatory increase of beta-cell function in the situation of insulin resistance
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